Dwayne Johnson's Rock- Hard Hercules Workout And Diet Plan. To play the son of Zeus in the upcoming blockbuster ''Hercules,'' Dwayne ''The Rock'' Johnson took his training to an entirely new level. In this interview, the movie icon reveals how you can build the body of a demigod. Many have tried in the past, and many have failed. Even for Johnson, with 2. But after spending the majority of his life in the gym training to be a professional football player, world- renowned pro wrestler, and one of Hollywood's highest- ever grossing movie stars, it was a labor he had all the weapons to overcome. Here, in his own words, Johnson reveals how he obtained the skin- stretching muscle he needed for his starring role as Hercules, and explains why nutrition is the most important facet of getting big. Hercules Official Trailer. Not so with Dwayne Johnson. That's why he was Hollywood's highest- grossing movie star of 2. If there's one thing life has taught the former national championship- winning footballer, it's that you only get what you're willing to put in. Madison's biographers generally agree little is known about the private life, including food likes/dislikes, of James Madison. Dolley Madison's lavish. Pink Shares Gym Selfie, Says She's 'Obese' by 'Regular Standards': 'Stay Off That Scale, Ladies!'. TODAY Parents is the premiere destination for parenting news, advice & community. Find the latest parenting trends and tips for your kids and family on TODAY.com. How Intermittent Fasting Stacks Up Among Obesity-Related Myths, Assumptions, and Evidence-Backed Facts. Get more consistent and regular movement into your life. More than 10,000 published studies confirm that sitting is an independent risk.That's why he dedicates so much of his time and energy to making sure he's at his physical peak when he steps onto the movie set. For 'Hercules,' it was a 2. G. I. Joe: Retaliation' it was about a 1. Pain & Gain' I wanted to come out looking bulky, big, and dangerous, so we adjusted accordingly. When You're Playing A Character Like The Son Of Zeus, You Only Get One Shot. When you're playing a character like the son of Zeus, you only get one shot. The intensity of the training was definitely up, as was the volume of training. I really wanted to make it the definitive version of Hercules. We wanted to make it darker in tone and we based it off Steve Moore and Admira Wijaya's 'Hercules: The Thracian Wars' graphic novel, which is pretty dark. The training, nutrition, fight choreography, weapons training, and stunt planning are all altered accordingly. I always go in committed and try to make it the best it can be. For 'Hercules,' I wanted to respect the mythology, or with . Joe movies, it was similar because it's already an established brand and already an established character. You've got to look the part. At the beginning of each morning, Johnson would complete a 5. When I'm filming, I do cardio and I lift before going to set. I train about six days a week, and even when I'm not filming I get up between 3 and 5 a. I love training when the sun is coming up because it allows me to put on my headphones and step off the crazy treadmill that is everyone's life. I have my headphones on and I'm listening to my music, and I'm 1. There's no wasted time or effort when it comes to me and the weights. When I hit the gym, I'm coming! Start studying OB TEST 1 PRACTICE. Learn vocabulary, terms, and more with flashcards, games, and other study tools. The Mediterranean Diet . To set the stage, here is a selection of titles from my local bookstore Need to plan a "decade" food event? This is a very doable project. Once you figure out what you want to accomplish, the rest will fall in place. I train hard, I come to kick ass. Generally, I'm always shooting or preparing to shoot for something, so that's why I like keeping my conditioning in a certain zone. But even if I wasn't it would launch my day. It makes me feels good. Lots of protein, good carbs, and veggies. Nutrition is so important, it can't be stressed enough. I'd eat a bunch of egg whites, filet mignon, chicken, fish, oatmeal, broccoli, asparagus, a baked potato, cream of rice, salad and complex carbs. All of this measured and weighed. I had carbs with every meal, so there was always a balance of proteins, carbohydrates, and fats. Eventually I hired an awesome conditioning coach who helped me. And when we get into our 4. I take glucosamine and chondroitin for my poor joints. Here's a man who has to accept and embrace his own myth. And that damn loincloth, there can be mishaps if you know what I mean- -when you're swinging about. TRAIN and Bodybuilding. Johnson's workout regimen. Remember to do an hour of cardio before you hit the weights. Reps. 43 sets, 2. Reps. 63 sets, 1. Reps. 73 sets, 2. Reps. Print. 13 sets, 1. Reps. 24 sets, 1. Reps. 34 sets, to failure. Reps. 84 sets, 2. Reps. 13. 3 sets, 2. Reps. 43 sets, 2. Reps. 63 sets, 1. Reps. 73 sets, 2. Reps. Can You Smell What The Rock Is Cookin'? Although Johnson didn't have anything in his diet quite like the blood of the Nemean lion Hercules had to kill, that didn't stop him from taking on the tough. The straight dope on cholesterol – Part VI - Previously, in Part I, Part II, Part III, Part IV and Part V of this series, we addressed these 7 concepts: #1 — What is cholesterol? It is not absorbed and is excreted by our gut (i. The reason this occurs is that CE not only has to be de- esterified, but it competes for absorption with the vastly larger amounts of UC supplied by the biliary route. Re- absorption of the cholesterol we synthesize in our body (i. That is, most of the cholesterol in our body was made by our body. The process of regulating cholesterol is very complex and multifaceted with multiple layers of control. You will discover that synthesis and absorption are very interrelated. Eating cholesterol has very little impact on the cholesterol levels in your body. This is a fact, not my opinion. Years ago the Canadian Guidelines removed the limitation of dietary cholesterol. The rest of the world, especially the United States, needs to catch up. Their cholesterol is trafficked back to the liver. The apo. A- I containing particles traffic cholesterol to steroidogenic tissues, adipocytes (a storage organ for cholesterol ester) and ultimately back to the liver, gut, or steroidogenic tissue. All lipoproteins are part of the human lipid transportation system and work harmoniously together to efficiently traffic lipids. As you are probably starting to appreciate, the trafficking pattern is highly complex and the lipoproteins constantly exchange their core and surface lipids. The measurement of cholesterol has undergone a dramatic evolution over the past 7. Currently, most people in the United States (and the world for that matter) undergo a “standard” lipid panel, which only directly measures TC, TG, and HDL- C. However, Lp(a) and apo. B containing lipoproteins play a role also, especially in the insulin resistant person. If you want to stop atherosclerosis, you must lower the LDL particle number. At first glance it would seem that patients with smaller LDL particles are at greater risk for atherosclerosis than patients with large LDL particles, all things equal.“A particle is a particle is a particle.” If you don’t know the number, you don’t know the risk. To address this question, however, one must look at changes in cardiovascular events or direct markers of atherosclerosis (e. IMT) while holding LDL- P constant and then again holding LDL size constant. This may explain the paucity of girlfriends in college. Let me use an example to illustrate the distinction between these terms. For one thing, we can’t do the “perfect” experiment. As you can see, conveniently, LDL- C values in mg/d. L are about 1. 0x off from LDL- P values in nmol/L. But this is not the case. Let’s look at these data parsed out another way. Only a minority of physicians would know that there is a segment of the population with elevated LDL- C who are at low risk! The same conclusion will be drawn from the next study. Let’s look at an even longer- term follow up study, below. So “high” implies above these values; “low” implies below these values. Populations where LDL- P and LDL- C discordance are even more prevalent. As I described above, the discordance between LDL- P and LDL- C is exacerbated in patients with metabolic syndrome. It takes many more cholesterol- depleted LDL particles than cholesterol- rich LDL particles to traffic a given cholesterol mass (i. L). Regardless of size it takes many more TG- rich LDL particles (which are necessarily cholesterol- depleted) to traffic a given cholesterol mass than TG- poor LDL particles. The solid red line shows perfect concordance between LDL- P and LDL- C. Now look again. When people argue with me about why it’s unnecessary to check LDL- P or apo. B because it’s much easier and cheaper to check LDL- C, I like to remind them of what Clint Eastwood would probably say in such a situation: “You’ve got to ask yourself one question: Do I feel lucky? Well, do ya, punk?”With respect to laboratory medicine, two markers that have a high correlation with a given outcome are concordant – they equally predict the same outcome. However, when the two tests do not correlate with each other they are said to be discordant. LDL- P (or apo. B) is the best predictor of adverse cardiac events, which has been documented repeatedly in every major cardiovascular risk study. LDL- C is only a good predictor of adverse cardiac events when it is concordant with LDL- P; otherwise it is a poor predictor of risk. There is no way of determining which individual patient may have discordant LDL- C and LDL- P without measuring both markers. Discordance between LDL- C and LDL- P is even greater in populations with metabolic syndrome, including patients with diabetes. Parents: Parenting News & Advice for Moms and Dadskellyclarkson/Instagram. Two- year- old River Rose gave the popular spread two messy thumbs- up in an Instagram video her.. TODAYThe U. N. There's surprising insight into.. TODAYAccording to a recent UNICEF study on well- being, children from the Netherlands are the happiest.. Courtesy of the Puma family. The Puma family thought daughter Tessa had a nasty flu, but it turned out to be a rare infection.. Zo. Laugh along with the funniest parents on Facebook.. Courtesy of Hoda Kotb. While Hoda Kotb hasn't been here in person since welcoming her baby girl, she called in Friday.. Shutterstock. 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Courtesy of Brandi Benner. Sophia was celebrating a big achievement with a new doll, and her mom never could.. Jill Simonian. My decision allowed me to feel physically independent, free of pain, and exponentially less exhausted.. Its Benefits and Role in Cancer Treatment. By Dr. Mercola. Could a ketogenic diet eventually be a ? Personally, I believe it's absolutely crucial, for whatever type of cancer you're trying to address, and hopefully some day it will be adopted as a first line of treatment. Additionally, low protein intake tends to minimize the m. TOR pathway that accelerates cell proliferation. It's a diet that will help optimize your weight and all chronic degenerative disease. Eating this way will help you convert from carb burning mode to fat burning. Thomas Seyfried is one of the leading pioneer academic researchers in promoting how to treat cancer nutritionally. He's been teaching neurogenetics and neurochemistry as it relates to cancer treatment at Yale University and Boston College for the past 2. Seyfried and his team worked on brain cancer and epilepsy in mice, one of his students suggested investigating whether or not a ketogenic diet might also be effective against tumors. And then we started to grow and substantially increase interest mainly through the efforts of Jim Abrahams. Jim started the Charlie Foundation for his son Charlie, who went through a near- death experience from seizures and was rescued using ketogenic diets. His colleague, Meryl Streep, the famous movie actress, became very involved in this. Now the ketogenic diet is receiving considerable attention in the epilepsy community as a first line of approach. Although this is still not widely accepted, I have to admit that the ketogenic diet is now recognized as an important component for the management of refractory seizures in children. Seyfried, the mechanism by which the ketogenic diet manages seizures is not nearly as clear as the way the ketogenic diet manages cancer. This is ironic considering that it's barely known, let alone applied, within oncology circles, while it's already a first line of treatment for epilepsy. In the case of cancer, it's well- established that it's the glucose reduction that kills the cancer cells. Cancer Is a Mitochondrial Metabolic Disease. Dr. Seyfried has developed a process called metabolic control analysis, which essentially analyzes the metabolic flux through different pathways that occurs when you transition your body from one major fuel source to another major fuel source, to maintain energy homeostasis in your body. Many believe or are under the impression that cancer is primarily a genetic disease, but Dr. Seyfried dispels such notions. But in order to do that, you have to present a massive counterargument against the gene theory of cancer. You can actually get normal tissues and sometimes a whole normal organism from the nucleus of a cancer cell. Now, if the tumors are being driven by driver genes – all these kinds of mutations and things that we hear about – how is it possible that all of this is changed when you place this cancer nucleus into the cytoplasm of a cell with normal mitochondria? The gene theory cannot address this. It clearly argues strongly against the concept that genes are driving this process. Most people inherit genes that prevent cancer. And those few genes that are inherited – the germ line like the BRCA1 mutations, B5. Seyfried, the mitochondria—the main power generators in your cells—are the central point in the origin of most cancers. Your mitochondria can be damaged not only by inherited mutations, thereby increasing your risk for a particular type of cancer, such as the BRCA1 and BRCA2 mutations that increase your risk of breast and ovarian cancer. They can also be damaged by environmental factors, such as toxins and radiation, both ionizing and non- ionizing. Over time, damage to your mitochondria can lead to dysfunction and tumor formation. The drugs that have been developed based on the genome projects have been largely ineffective in providing long- term care and are associated with toxic effects. As long as the field continues to focus on that part of the disease, which is a downstream epiphenomenon, there will be no major advances in the field simply because that's not the relevant aspect of the disease. These factors are also crucial in order to prevent cancer in the first place. In 1. 93. 1, the Nobel Prize was awarded to German researcher Dr. Otto Warburg, who discovered that cancer cells have a fundamentally different energy metabolism compared to healthy cells, and that malignant tumors tend to feed on sugar. More recently, researchers discovered that while cancer cells feed on both glucose and fructose, pancreatic tumor cells use fructose specifically to divide and proliferate. Seyfried's work confirms that sugar is the primary fuel for cancer, and that by restricting sugar and providing an alternate fuel, namely fat, you can dramatically reduce the rate of growth of cancer. We know that glucose is the primary fuel for fermentation. Fermentation becomes a primary energy- generating process in the tumor cell. By targeting the fuel for that process, we then have the capability of potentially managing the disease. Seyfried suggests is a low- carb, low to moderate protein, high- fat diet, which will effectively lower your blood sugar. This is an easily measurable parameter that you can check using a diabetic blood glucose meter. This type of diet, called a ketogenic diet, will also elevate ketone bodies, as fat is metabolized to ketones that your body can burn in the absence of food. When combined with calorie restriction, the end result will put your body in a metabolic state that is inhospitable to cancer cells. So the ketogenic diet represents an elegant, non- toxic way to target and marginalize tumor cells. It also allows you to dramatically lower your glucose levels, as the ketones will protect your body against any hypoglycemia that might otherwise be induced by carb restriction. At the same time, the tumor cells are now marginalized and under tremendous metabolic stress. It's a whole body therapy—you need to bring the whole body into this metabolic state. If it's done right and implemented right, it has powerful therapeutic benefits on the majority of people who suffer from various kinds of cancers. Because all cancers have primarily the same metabolic defect. Seyfried uses ketones and glucose as the measures of this new metabolic state. The parameters associated with an ideal state are ketone levels equal to or higher than the glucose level in your blood. But in a fasted or therapeutic state, this ratio is actually reversed. Ketones can actually become higher than glucose. It's this state that now brings the body into this new physiology. Generally speaking, a fasting glucose under 1. Seyfried recommends getting your glucose down to a steady level of about 5. HALF of what's conventionally considered . As many pharmacies might not stock the meter (bar code #, 9. Abbott directly (1- 8. Seyfried, the Precision Xtra seems the most accurate of all the ones he's used. Seyfried therefore recommends measuring your blood ketones every few days rather than 3x/day for blood glucose. Although urine ketone measurement is a cheap way to assess ketones, urine ketone levels are not always indicative of blood ketone levels. It is best if you can measure ketones from both blood and urine. We have knowledge of how to do this. We have patients willing to do it. But we lack professionals that are trained or even understand the concepts of how to implement these kinds of approaches. Seyfried's book, Cancer as Metabolic Disease, which is available on Amazon. He's also published a couple of papers 1,2 that outline the guidelines and treatment strategies for cancer patients. One caveat to consider is your use of medications, as you need to know what the adverse effects might be if you use a medication at a particular dosage along with this kind of metabolic therapy. The Importance of Intermittent Fasting. In my experience, the vast majority of people are adapted to burning carbs as their primary fuel, as opposed to burning fat. One of the most effective strategies I know of to become a fat burner is to restrict your eating to within a six- to eight- hour window, which means you're fasting for about 1. This upregulates the enzymes that are designed to burn fat as a fuel, and downregulates the glucose enzymes. This kind of intermittent fasting plan can be a useful modality to help you make the transition to a ketogenic diet. Basically, the child is given a 2. And then the ketogenic diet is introduced in relatively measured and small amounts. Intermittent fasting is actually a very strong component of the approach. A three- day fast is uncomfortable, but it's certainly doable. It gets your body into a new metabolic state, and then you can apply these therapies. The hardest part, I think, of this fasting is the first three to four days, depending on the individual and how many times they've done this. That's basically trying to break your addiction to glucose. The removal of glucose from the brain elicits the same kind of problems or events as you would if you were addicted to drugs, alcohol, or something like this. You get malaise, headaches, nausea, lightheadedness. You get all the kinds of physiological effects that you would get from withdrawal of any addicting substance. I look at glucose as an addictive substance. It's an addictive metabolite. Your brain is comforted by having glucose; your body is comforted. And when you break that glucose addiction, you have these particular feelings. Fasting certainly has remarkable health benefits to the body: strengthening the mitochondria network system within the cells of your body. As long as the mitochondria of your cells remain healthy and functional, it's very unlikely that cancer can develop under these particular states. You begin by not eating for three hours before you go to bed, and then gradually extend the time you eat breakfast until you have skipped breakfast entirely and your first meal of the day is at lunch time.
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